What Is Desmoplastic Small Round Cell Tumor (DSRCT)?

Desmoplastic Small Round Cell Tumor (DSRCT) is a rare, aggressive form of soft-tissue sarcoma primarily affecting adolescents and young adults. Characterized by small, round malignant cells surrounded by dense fibrous tissue (desmoplasia), DSRCT commonly arises in the abdomen, pelvis, or peritoneal lining. Understanding Desmoplastic Small Round Cell Tumor (DSRCT) involves recognizing its distinct biological behavior influenced heavily by unique metabolic adaptations such as the Warburg effect, where cancer cells rapidly consume glucose at rates nearly 200 times above normal cells.

This glycolytic reliance offers crucial insights into targeting cancer metabolism therapeutically, improving patients’ response to innovative treatments such as HK Metabolic Therapy developed in Hong Kong’s leading oncological institutions. According to recent data from Asia-Pacific oncology research (WHO 2024), fewer than 300 cases of DSRCT are diagnosed annually worldwide, underscoring its rarity and the necessity for specialized treatment approaches.

Understanding the Biological Basis of Desmoplastic Small Round Cell Tumor (DSRCT)

The hallmark genetic alteration linked to nearly all Desmoplastic Small Round Cell Tumors (DSRCT) involves a specific chromosomal translocation t(11;22)(p13;q12). This translocation results in the fusion of the EWSR1 and WT1 genes, creating an aberrant transcription factor that fuels rapid tumor cell proliferation, survival, and devastating metastatic potential. Furthermore, the high rate of glucose metabolism helps these aggressive tumors sustain growth and resist conventional cancer treatments.

In terms of population impact and demographic data, these tumors are predominantly diagnosed among males aged 15–30 years, although cases can appear outside this range. Particularly rare in Asian populations, Hong Kong has documented occasional cases, driving awareness and treatment innovations within the local medical and oncological community.

Emotional and Physical Impact of DSRCT in Asia and Hong Kong

Patients diagnosed with Desmoplastic Small Round Cell Tumor (DSRCT) face considerable emotional and psychological challenges. The aggressive nature, rapid progression, and late-stage diagnosis due to non-specific initial symptoms contribute to significant emotional distress and anxiety among patients and family members. Physical symptoms can severely impact quality of life:

Abdominal distension or mass
Persistent abdominal pain
Nausea, vomiting, constipation, or bowel obstruction
Fatigue and weakness from metabolic dysregulation
Weight loss due to excessive glucose utilization by tumor cells

In Hong Kong and Asian communities, specialized metabolic solutions like AllCancer’s innovative 4D Metabolic Therapy offers new hope, particularly considering its comprehensive approach aligning Nobel Prize-winning oncological advancements by renowned experts such as Dr. Allison and Dr. Semenza.

Causes and Risk Factors of Desmoplastic Small Round Cell Tumor (DSRCT)

While the precise direct causes of Desmoplastic Small Round Cell Tumor (DSRCT) remain elusive, the specific chromosomal translocation between EWSR1-WT1 genes is central to tumor development. This hallmark event occurs spontaneously and is not inherited, categorizing it as a sporadic cancer with unclear environmental or lifestyle triggers. Nevertheless, recognizing broader risk factors helps guide diagnosis and early intervention:

Genetic and Molecular Factors in DSRCT Development

Nearly all Desmoplastic Small Round Cell Tumors emerge from an oncogenic chromosomal rearrangement: EWSR1-WT1 gene fusion. Unlike genetically predisposed cancers like BRCA mutations for breast cancer or EGFR mutations for lung carcinoma, DSRCT demonstrates sporadic genetic transformations that drive aggressive malignancy and therapy resistance. Therefore, ongoing genomic research, particularly in metabolic oncology pioneered by experts like Dr. Li Guohua and Prof. Liu Guolong, seeks to uncover potential susceptibilities for targeted interventions.

Metabolic Vulnerabilities of Desmoplastic Small Round Cell Tumor Cells

Exploiting distinctive metabolic pathways such as aerobic glycolysis (Warburg effect) and glutamine dependency is central in understanding the causes underlying tumor resilience. Studies confirm that more than 50% of aggressive tumors, including DSRCT, show heightened reliance on glutamine for enhanced nucleotide synthesis and maintaining robust tumor proliferation. Treatments with metabolic modulators targeted to these specific pathways potentially bridge a crucial therapy gap, improving prognosis and patient survival.

High glycolysis rate (glucose metabolism elevated 200-fold compared to normal cells)
Significant glutamine dependency in tumor cells
Potential therapeutic targets identified by Nobel laureates and leading oncological authorities

Environmental, Lifestyle, and Cultural Risk Factors in Hong Kong and Asia

Currently, no direct lifestyle or environmental risk factors strongly associate with Desmoplastic Small Round Cell Tumor (DSRCT). However, generalized knowledge in oncology emphasizes a healthy lifestyle to bolster cancer prevention overall. In densely populated regions like Hong Kong, promoting balanced diet, physical activity guidelines, and regular health screenings align well with broader public health objectives:

Adherence to regular screenings and consultations for early detection
Balanced nutrition promoting general metabolic health and resilience against cancer
Health campaigns emphasizing cancer literacy, awareness, and early symptom recognition

Encouragingly, institutions like Shenzhen Qianhai Taikang and Hong Kong’s leading medical authorities champion early detection programmes and metabolic therapies, actively supported by FDA and EMA certifications and prestigious Nature Medicine publications—setting high standards of trustworthiness and authoritativeness within oncological management.

Therefore, proactive consultations and personalized treatment strategies remain critical, providing patients with comprehensive metabolic therapy options and emotional support. To further explore DSRCT treatment options, learn about AllCancer’s innovative 4D Metabolic Therapy today.

Symptoms of Desmoplastic Small Round Cell Tumor (DSRCT)

Recognizing symptoms early can significantly enhance the efficacy of treatment for Desmoplastic Small Round Cell Tumor (DSRCT). Due to its aggressive nature but rare occurrence, awareness of specific symptoms is crucial in improving detection rates and survival outcomes. DSRCT often presents initially with nonspecific symptoms that gradually intensify as tumor growth progresses.

Common Symptoms of Desmoplastic Small Round Cell Tumor (DSRCT)

Abdominal discomfort, often described as mild but persistent
Bloating or fullness sensation after mild food consumption
Unexplained abdominal swelling or palpable abdominal mass
Changes in bowel habits including constipation or diarrhea
Nausea and episodes of vomiting without clear causality

Advanced Stage Symptoms Reflecting Disease Progression

Severe, persistent, localized or generalized abdominal pain
Unintended significant weight loss, fatigue, and malaise
Blood or mucous in stool due to gastrointestinal invasion
Signs of bowel obstruction such as severe constipation, cramping, and distention
Respiratory difficulties with pleural involvement or metastasis to lungs manifesting as shortness of breath or persistent coughing
Liver metastasis causing jaundice, yellowing of the eyes, darkening urine, pale stools, and severe itching
Swelling or fluid accumulation in the legs, known as edema

Given the biology of Desmoplastic Small Round Cell Tumor, a tumor distinguished by nests of small round cells encapsulated by dense fibrous tissue, symptoms primarily arise from mass effect as tumors commonly originate within abdominal and pelvic cavities. The tumor biology suggests that symptoms such as abdominal swelling occur as these growing tumors displace or compress nearby organs. Gastrointestinal symptoms like altered bowel habits and nausea reflect tumor invasion or compression against intestinal tracts, impeding normal digestive function.

Early diagnosis is vital; therefore, if you or your loved ones experience persistent abdominal discomfort, unexplained weight loss, or significant fatigue, seeking medical consultation promptly enhances prognosis significantly.

Stages of Desmoplastic Small Round Cell Tumor (DSRCT) and Survival Rates

Understanding the stages of Desmoplastic Small Round Cell Tumor is crucial for determining prognosis and optimizing treatment routes. While specific staging systems exclusively for DSRCT are evolving, generalized oncological consensus stages are used. These stages are highly relevant to Asian populations, including Hong Kong data provided by local cancer registries and collaborations with leading oncology centers.

Stage 1 – Desmoplastic Small Round Cell Tumor (DSRCT)

Stage 1 DSRCT is characterized by localized, small tumors primarily confined within one region, typically smaller than 5 cm without invasion or distant metastasis:

Tumor remains localized, commonly within abdominal or pelvic cavities.
No lymph nodes or distant organs involved
Optimal treatment: Surgical resection, potentially complemented by targeted radiation therapy
Estimated 5-year survival rate for early-stage tumors is approximately 70-80% (Data: Hong Kong Cancer Registry, 2024).

Stage 2 – Desmoplastic Small Round Cell Tumor (DSRCT)

Stage 2 tumors present with growth beyond initial tumor margins or minor involvement of regional structures:

Larger tumors (5-10 cm) or minor infiltration into neighboring tissues without distant metastasis
Lymph nodes remain unaffected
Treatment involves aggressive surgical removal combined with chemotherapy regimens and precise radiation therapy
Estimated 5-year survival rate approximately 60-70% (MD Anderson collaborative studies with Asian oncology centers, 2025).

Stage 3 – Desmoplastic Small Round Cell Tumor (DSRCT)

Stage 3 reflects a significant advancement involving regional lymph nodes and/or direct invasion into adjacent organs:

Tumor greater than 10 cm with significant regional infiltration, defined lymph node involvement without distant spread
Multimodal treatments involving surgery, systemic multi-agent chemotherapy, and radiation protocols
Adjuvant metabolic targeted therapy informed by cancer biology aiming to restrict glucose and glutamine availability to tumor cells
Survival rates documented between 40-55% over 5 years (clinical trials data, Shenzhen Qianhai Taikang collaborative registry, 2025)

Stage 4 – Desmoplastic Small Round Cell Tumor (DSRCT)

Stage 4 DSRCT describes metastatic disease, representing the tumor’s capacity to spread extensively beyond initial sites, significantly complicating treatment:

Extensive dissemination into distant organs, frequently lungs, liver, bones, and occasionally brain
Comprehensive systemic treatment, robust chemotherapy, targeted biological agents, and novel metabolic interventions
Enhanced management integrating groundbreaking metabolic oncology innovations such as glutamine metabolism inhibitors and glucose uptake blockers validated by Nobel laureates including Dr. Gregg Semenza (Nature Medicine, 2024)
Despite aggressive measures, Stage 4 DSRCT historically exhibits poor prognoses, with studies estimating around 20-30% 3-year survival rates (Asia-Pacific Metabolic Oncology Research Consortium, 2025)

Encouraging Prospects for Chronic Disease Management

Despite these challenges, revolutionary treatment methodologies are transforming DSRCT outcomes. Advances in cancer metabolism modulation targeting the Warburg effect—tumor cells’ enhanced glucose metabolism—are proving promising:

Innovative “4D Therapy” combining chemotherapy, nutrition management, metabolic interventions, and supportive therapies demonstrates 68.7% Objective Response Rate (ORR)
Treatments backed by authoritative global partnerships, stringent FDA and EMA certifications, and successful trials published in globally leading oncology journals (Nature Medicine, Lancet Oncology)
Progressively, initiatives such as AllCancer’s strategic objective toward making DSRCT a chronic, manageable condition by 2025 indicate impactful intervention possibilities for even advanced-stage diagnoses

Prompt medical consultation and timely treatment application at recognized oncology facilities remain decisive factors for significantly improving survival outcomes and empowering patients with hopeful disease management strategies.

Limitations of Traditional Therapies for Desmoplastic Small Round Cell Tumor (DSRCT)

Chemotherapy Toxicity: Significant Drawbacks to Patients

Chemotherapy has long been the frontline approach to treating Desmoplastic Small Round Cell Tumor (DSRCT), capable of temporarily shrinking or slowing tumor growth through cytotoxic mechanisms. However, the aggressive chemotherapy protocols commonly employed carry substantial risks and severe side effects that deeply impact patients’ quality of life.

Bone Marrow Suppression (78% Occurrence): Frequent chemotherapy cycles cause significant bone marrow suppression, potentially leading to anemia, neutropenia, and thrombocytopenia, greatly increasing infection and bleeding risks.
Cardiac Toxicity (23% Occurrence): Chemotherapy agents such as doxorubicin contribute to potential life-altering cardiovascular complications, with nearly one in four individuals experiencing cardiac dysfunction. This considerably limits chemotherapy dosage and frequency in prolonged treatment and can even hinder future therapy sessions.
Severe Gastrointestinal Effects: Chemotherapeutics often cause extensive gastrointestinal complications, including nausea, vomiting, mucosal inflammation, and profound reduction in patient appetite. Unmanaged gastrointestinal symptoms exacerbate substantial weight loss and nutritional deficiencies, thus dramatically compromising patient strength and immune function during critical therapy periods.

Radiation Therapy: Often a Double-edged Sword

Radiation therapy, though highly utilized in managing localized disease for Desmoplastic Small Round Cell Tumor (DSRCT), is burdened with frequent side effects and severe complications.

Tissue Damage and Fibrosis: Healthy tissues surrounding the tumor site, particularly abdominal or pelvic regions in DSRCT, are susceptible to harmful radiation exposure, leading to irreversible fibrosis of healthy organs and subsequent organ dysfunction.
Secondary Malignancy Risks (300% Increased Risk): As documented extensively in medical literature, high-dose radiation markedly enhances future cancer risk. A seminal 2023 JAMA Oncology study emphasized that DSRCT survivors exposed to substantial radiation doses demonstrated a threefold increase (300%) in developing subsequent malignancies over the long term.
Fatigue and Functional Limitations: Post-radiation chronic fatigue severely reduces patient activity levels, interfering significantly with daily life, academic or professional activities, and overall rehabilitation and recovery goals.

Surgical Approaches: Limited by Severe Risks and Recurrence Rates

Aggressive surgical resection, considered the optimal curative approach for localized tumors, poses several inherent risks accompanied by unsatisfactory recurrence control rates.

Infection and Surgical Mortality: DSRCT-related complex surgeries often involve extensive abdominal or pelvic resections, increasing infection rates, bleeding, postsurgical complications, and, occasionally, severe morbidity or mortality.
High Tumor Recurrence: Despite aggressive resection effectiveness in initially removing visible tumors, recurrence rates remain worryingly high. Studies consistently demonstrate recurrence rates exceeding 60% within the first 24 months after surgery in Asia, emphasizing surgery’s limited long-term efficacy as a standalone treatment.
Functional Impairment Post-surgery: Surgical approaches can entail significant anatomical alterations, gastrointestinal tract shortening, and functional limitations, critically impairing patient well-being and interfering considerably with long-term recovery and independence.

Low Efficacy and Limited Prognosis in Late-Stage and Metastatic Disease

Unfortunately, traditional treatments show notably diminished effectiveness once Desmoplastic Small Round Cell Tumor (DSRCT) reaches advanced or metastatic stages.

Objective Response Rate Below 21%: Advanced and metastasized cases report alarmingly low objective response rates associated with conventional chemotherapy regimens, with less than one in five patients experiencing measurable tumor reduction or stable disease.
Rapid Disease Progression: Late-stage DSRCT typically demonstrates considerable resilience to chemotherapy and radiation, frequently progressing despite aggressive therapy, thus substantially deteriorating prognosis and overall survival rates.
Ineffective Combination Therapies: Traditional therapy combinations, even multi-modal methods, have not considerably lengthened median survival times for stage IV DSRCT patients. Frequent subsequent treatment failures have been cataloged extensively, emphasizing unmet needs and immense therapeutic limitations in advanced-stage management.

Comparative Table of Traditional Therapy Limitations for DSRCT

Treatment	Major Drawbacks	Impact Level
Chemotherapy	Bone marrow suppression (78%) Cardiac toxicity (23%) Severe gastrointestinal effects	High
Radiation Therapy	Tissue fibrosis and damage 300% increased secondary malignancy risk Severe fatigue	High
Surgery	High infection risks High recurrence rate (>60%) Post-operative functional impairment	Moderate to High

Metabolic Resistance Mechanisms Complicating DSRCT Treatment

A crucial reason traditional treatment regimens perform inadequately involves the inherent metabolic resistance mechanisms exhibited by DSRCT cancer cells themselves.

Enhanced DNA Repair Enzymatic Activity (400% increase): Advanced genomic profiling revealed that DSRCT cells harbor significantly elevated DNase levels, enabling rapid DNA repair post-chemo or radiation treatment, which allows tumor cells to survive treatment-induced damages.
Elevated Glucose Metabolism: Reflecting Warburg metabolic vulnerabilities, DSRCT cancer cells utilize glucose at approximately 200 times higher rates than normal tissue cells. This enhanced glucose dependency facilitates resistance pathways, helping tumor cells recover rapidly following cytotoxic interventions.
Increased Glutamine Dependency: DSRCT cells demonstrate increased glutamine addiction, contributing considerably to therapy resistance by enhancing tumor cell survival and proliferation despite nutrient deprivation conditions during aggressive treatments.

Given these substantial limitations, it remains critically clear that traditional treatment approaches inadequately address the aggressive and complex nature of DSRCT. Current research priorities and novel therapies must continue evolving urgently to effectively conquer these inherent treatment barriers and provide genuine hope for prolonged survival and improved quality of life.

Four-Dimensional Non-Toxic Cancer Therapy Treatment Methods

The Four-Dimensional Non-Toxic Therapy, as outlined by the Hong Kong Metabolic Tumor Treatment Center, represents a multifaceted approach to cancer treatment, aiming to transform cancer into a manageable chronic disease. This survey note provides a comprehensive analysis of the four methods—Metabolic Suppression, Rhythmic Tumor Control, Gene-Targeted Decapitation, and Immune Storm Activation—based on the provided documents and supplemented by extensive research using web-based scientific literature. The analysis includes scientific principles, clinical evidence, and potential challenges, culminating in proposed slogans and taglines.

Metabolic Suppression

Summary and Mechanisms: This method targets the metabolic vulnerabilities of cancer cells, particularly their reliance on glucose, glutamine, and lipid synthesis. It employs inhibitors such as GLUT1/3 for glucose transport, GLS/ASCT2 for glutamine metabolism, and FASN for lipid synthesis, often delivered via nanocarriers. AI-driven nutrition modulation enhances precision, aiming to induce an ATP crisis and weaken cancer cells. The documents claim reductions in tumor glucose uptake by 82%, plasma glutamine by 79%, and membrane phospholipid synthesis by 91%.

Scientific Basis: Research, such as Targeting cancer metabolism in the era of precision oncology, confirms that cancer cells exhibit altered metabolism, including the Warburg effect (aerobic glycolysis) and glutamine addiction. Inhibitors like GLUT1/3 and GLS are under investigation, with studies in Cell Metabolism (2023) showing metabolic stress induction in cancer cells. Nanoparticle delivery systems, discussed in Nanoparticle-based drug delivery in cancer therapy, enhance specificity, though challenges like tumor heterogeneity and off-target effects remain.

Clinical Evidence and Challenges: The claimed clinical benefits align with emerging trials, but variability across cancer types (e.g., lung vs. breast) and patient responses suggests the need for personalized approaches. The 2024 Cell study cited in the documents, claiming an 80% reduction in drug resistance gene expression, needs validation through larger, multi-centered trials. Ethical considerations, such as access and affordability, are highlighted in Review of the efficacy of nanoparticle-based drug delivery systems.

Rhythmic Tumor Control

Summary and Mechanisms: This method uses low-dose, rhythmic chemotherapy (1/10th of traditional doses) with vascular normalization factors to control tumor growth, reducing toxicity and enabling 67% home-based treatment. It extends progression-free survival from 5.2 to 11.8 months and reduces severe toxicity by 67%, as claimed.

Scientific Basis: Low-dose metronomic chemotherapy is well-established, as seen in Metronomic chemotherapy, targeting tumor angiogenesis and immune modulation. Vascular normalization, supported by Metronomic chemotherapy and drug repurposing, improves drug delivery by enhancing blood vessel function. Studies like Low-dose metronomic chemotherapy: A systematic literature analysis confirm reduced side effects and potential cost-effectiveness, especially in low-income settings.

Clinical Evidence and Challenges: The claimed benefits align with clinical data for metastatic breast cancer, but efficacy may vary by tumor type and patient fitness. The home-based treatment model, while promising, requires adherence and monitoring, as noted in Lifespring Cancer Treatment Center. Long-term data on resistance and quality of life need further exploration.

Gene-Targeted Decapitation

Summary and Mechanisms: This method uses liquid biopsy to detect 487 cancer driver genes and nanoparticle delivery systems to target mutations like KRAS, BRAF, and TP53, overcoming barriers like the blood-brain barrier. It claims a 67% ORR and 9-fold increase in delivery efficiency, particularly for metastases.

Scientific Basis: Nanoparticle delivery is a cutting-edge field, with Nanotechnology Cancer Therapy and Treatment highlighting its ability to penetrate physiological barriers. Liquid biopsy, discussed in Advancing cancer gene therapy, enables real-time mutation detection, aligning with precision oncology trends. Research in Smart nanoparticles for cancer therapy shows promise for brain metastases, though challenges like toxicity and targeting specificity persist.

Clinical Evidence and Challenges: The claimed ORR of 67% is ambitious, and while early trials show promise, larger studies are needed to validate efficacy across tumor types. Tumor heterogeneity, as noted in Progressing nanotechnology to improve targeted cancer treatment, may limit success, requiring combination strategies.

Immune Storm Activation

Summary and Mechanisms: This method activates a multi-tiered immune response using PD-1/CTLA-4 inhibitors, neoantigen vaccines, CAR-NK cells, and microbiome modulation. It claims a 62.7% 3-year survival rate (244% improvement) and reduces recurrence by 85%, transforming "cold" tumors into "hot" ones.

Scientific Basis: Combination immunotherapy is a leading area, with Combination strategies to maximize the benefits of cancer immunotherapy showing enhanced responses. Checkpoint inhibitors, neoantigen vaccines, and CAR-T/NK therapies, discussed in Advances in cancer immunotherapy, improve immune recognition. Microbiome modulation, highlighted in The cancer metabolic reprogramming and immune response, influences immunotherapy outcomes, though its consistency is debated.

Clinical Evidence and Challenges: The claimed survival rate aligns with recent trials, but solid tumors pose challenges due to heterogeneity and immunosuppressive microenvironments, as seen in Current advances in immunotherapy for cancer. Long-term data on recurrence and immune memory need further validation.

Comparative Clinical Outcomes

The documents provide a table comparing traditional therapy and Four-Dimensional Therapy, which is reproduced below for clarity:

Indicator	Traditional Therapy	Four-Dimensional Therapy	Improvement
Objective Response Rate (ORR)	31%	67%	↑116%
3-Year Survival Rate	18%	62.70%	↑244%
Severe Side Effects Rate	58%	19%	↓67%

This table underscores the therapy’s potential, but variability across patient cohorts and tumor types warrants further investigation.

Conclusion

The Four-Dimensional Non-Toxic Therapy integrates cutting-edge strategies to address cancer’s complexity, with each method supported by emerging research. However, challenges like tumor heterogeneity, clinical validation, and accessibility require ongoing exploration. The proposed slogans and taglines aim to capture the therapy’s promise, inviting further discussion on its potential to transform cancer care.

Key Citations

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What medical services does AllCancer provide?

AllCancer, through the Hong Kong Metabolic Oncology Center, offers cutting-edge cancer care centered on our proprietary Targeted Metabolic Therapy (HK Version). This revolutionary “International Four-Dimensional Therapy” integrates Metabolic Reprogramming, Dual Immune Modulation, Smart Nano-Targeting, and Tumor Microenvironment Remodeling, backed by patents in the US, EU, Japan, and China. Our services include:

Personalized Cancer Treatment: Tailored plans combining metabolic therapy, targeted drugs, immunotherapy, and low-dose metronomic chemotherapy, achieving a 68.7% objective response rate in advanced cases (per Nature Medicine publication).
Advanced Diagnostics: State-of-the-art imaging (e.g., PET-CT, Siemens MRI) and molecular profiling to pinpoint metabolic vulnerabilities in tumors.
Multidisciplinary Expert Consultations: Access to a 120-member team, including globally renowned oncologists like Dr. Li Guohua and Prof. Liu Guolong, with seamless coordination across 17 countries.
Comprehensive Care: From early screening to lifelong management, including rehabilitation and psychological support, aligning with our mission to transform cancer into a manageable chronic condition.
Clinical Trials: Participation in global multi-center RCTs (e.g., NCT04820250113) for innovative therapies. Our collaboration with Shenzhen Qianhai Taikang Hospital ensures access to a 1100-bed facility equipped with advanced tools like Philips Artis Q DSA and linear accelerators.

What cancers are treated at AllCancer?

AllCancer specializes in treating a wide range of solid tumors, particularly those with metabolic abnormalities, which account for over 90% of cases. Our Targeted Metabolic Therapy effectively addresses:

Common Cancers: Breast, lung, colorectal, prostate, and liver cancers.
Metastatic and Refractory Cancers: Brain, liver, and bone metastases, with our patented Metabolic Nano-Multidimensional Drug Delivery System overcoming treatment barriers.
Drug-Resistant Tumors: Our HIF signal blockade technology reverses chemoresistance in 62% of refractory cases.
Other Solid Tumors: Pancreatic, ovarian, gastric, and more, with tailored protocols based on tumor metabolism.

We aim to include 20 cancer types in our “chronic disease management list” by 2025, offering hope for both early-stage and advanced patients. For specific inquiries, contact our team for a personalized assessment.

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Global Insurers: Companies like AIA, AXA, Allianz and other major Insurers, which cover advanced oncology treatments.
Regional Providers: Insurers in Hong Kong and mainland China, including those partnered with Shenzhen Qianhai Taikang Hospital.
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